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浏览/检索结果: 共5条，第1-5条 帮助 限定条件 专题：中南大学    第一署名单位    第一作者单位    通讯作者单位     已选(0)清除 条数/页：5101520253035404550556065707580859095100   排序方式：请选择作者升序作者降序题名升序题名降序发表日期升序发表日期降序提交时间升序提交时间降序 BET Bromodomain Suppression Inhibits VEGF-induced Angiogenesis and Vascular Permeability by Blocking VEGFR2-mediated Activation of PAK1 and eNOS 期刊论文 SCIENTIFIC REPORTS, 2016, 卷号: 6, 页码: 23770 作者:  Huang, Mingcheng;  Qiu, Qian;  Xiao, Youjun;  Zeng, Shan;  Zhan, Mingying 收藏  |  浏览/下载：9/0  |  提交时间：2019/12/03 Adiponectin in Fresh Frozen Plasma Contributes to Restoration of Vascular Barrier Function After Hemorrhagic Shock. 期刊论文 Shock (Augusta, Ga.), 2016, 卷号: 45, 期号: 1, 页码: 50-54 作者:  Deng, Xiyun;  Cao, Yanna;  Huby, Maria P.;  Duan, Chaojun;  Baer, Lisa 收藏  |  浏览/下载：10/0  |  提交时间：2019/12/03 Endothelial cell hyperpermeability  human subjects  immunodepletion  mouse model  pulmonary microvascular permeability   Nitric Oxide Increases Arterial Endotheial Permeability through Mediating VE-Cadherin Expression during Arteriogenesis 期刊论文 PLOS ONE, 2015, 卷号: 10, 期号: 7, 页码: e0127931 作者:  Yang, Baolin;  Cai, Baizhen;  Deng, Panyue*;  Wu, Xiaoqiong;  Guan, Yinglu 收藏  |  浏览/下载：3/0  |  提交时间：2019/12/03 Vascular permeability,Nitric oxide,Femoral arteries,Macrophages,Endothelial cells,Quantitative analysis,Immunostaining,Monocytes   NMDA Receptor Antagonist Attenuates Bleomycin-Induced Acute Lung Injury 期刊论文 PLOS ONE, 2015, 卷号: 10, 期号: 5, 页码: e0125873 作者:  Li, Yang;  Liu, Yong;  Peng, XiangPing;  Liu, Wei;  Zhao, FeiYan 收藏  |  浏览/下载：8/0  |  提交时间：2019/12/03 Neutrophils,Inflammation,Glutamate,Cytokines,Mouse models,Central nervous system,Vascular permeability,Oxidative stress   TNF-alpha mediated increase of HIF-1alpha inhibits VASP expression, which reduces alveolar-capillary barrier function during acute lung injury (ALI). 期刊论文 PLOS ONE, 2014, 卷号: 9, 期号: 7, 页码: e102967 作者:  Tang, Mengjie;  Tian, Yihao;  Li, Doulin;  Lv, Jiawei;  Li, Qun 收藏  |  浏览/下载：4/0  |  提交时间：2019/12/03 Permeability,Inflammation,Mouse models,Vascular permeability,Gene expression,Protein expression,Tight junctions,Cytokines