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The inhibition of MyD88 and TRIF signaling serve equivalent roles in attenuating myocardial deterioration due to acute severe inflammation
Ouyang, Min-Zhi; Zhou, Dan; Zhu, Yun; Zhang, Ming*; Li, Ling(李玲)
刊名International journal of molecular medicine
2018
卷号41期号:1页码:399-408
关键词myeloid differentiation factor 88 signaling Toll or interleukin-1 receptor-domain-containing adaptor-inducing interferon- signaling severe sepsis myocardial dysfunction inflammatory response apoptosis
ISSN号1107-3756
DOI10.3892/ijmm.2017.3239
URL标识查看原文
WOS记录号WOS:000423432100042
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/5708803
专题湖南中医药大学
作者单位1.[Ouyang, Min-Zhi
2.Zhou, Dan
3.Zhu, Yun
4.Zhang, Ming] Cent S Univ, Dept Ultrasound Diag, Xiangya Hosp 2, 139 Renmin Rd, Changsha 410011, Hunan, Peoples R China.
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GB/T 7714
Ouyang, Min-Zhi,Zhou, Dan,Zhu, Yun,et al. The inhibition of MyD88 and TRIF signaling serve equivalent roles in attenuating myocardial deterioration due to acute severe inflammation[J]. International journal of molecular medicine,2018,41(1):399-408.
APA Ouyang, Min-Zhi,Zhou, Dan,Zhu, Yun,Zhang, Ming*,&Li, Ling.(2018).The inhibition of MyD88 and TRIF signaling serve equivalent roles in attenuating myocardial deterioration due to acute severe inflammation.International journal of molecular medicine,41(1),399-408.
MLA Ouyang, Min-Zhi,et al."The inhibition of MyD88 and TRIF signaling serve equivalent roles in attenuating myocardial deterioration due to acute severe inflammation".International journal of molecular medicine 41.1(2018):399-408.
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