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	E2F2 directly regulates the STAT1 and PI3K/AKT/NF-κB pathways to exacerbate the inflammatory phenotype in rheumatoid arthritis synovial fibroblasts and mouse embryonic fibroblasts
	Wang S.; Wang L.; Wu C.; Sun S.; Pan J.-H.
刊名	Arthritis Research and Therapy
	2018
卷号	20期号:1
关键词	Cytokine E2F2 Inflammation Rheumatoid arthritis Synovial fibroblast
DOI	10.1186/s13075-018-1713-x
URL标识	查看原文
公开日期	[db:dc_date_available]
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/4568136
专题	山东大学
作者单位	Medical and Life Science College, University of Jinan, Jinan, 250062, China, Shandong Medicinal Biotechnology Centre, Jingshi Road, Jinan, 2
推荐引用方式 GB/T 7714	Wang S.,Wang L.,Wu C.,et al. E2F2 directly regulates the STAT1 and PI3K/AKT/NF-κB pathways to exacerbate the inflammatory phenotype in rheumatoid arthritis synovial fibroblasts and mouse embryonic fibroblasts[J]. Arthritis Research and Therapy,2018,20(1).
APA	Wang S.,Wang L.,Wu C.,Sun S.,&Pan J.-H..(2018).E2F2 directly regulates the STAT1 and PI3K/AKT/NF-κB pathways to exacerbate the inflammatory phenotype in rheumatoid arthritis synovial fibroblasts and mouse embryonic fibroblasts.Arthritis Research and Therapy,20(1).
MLA	Wang S.,et al."E2F2 directly regulates the STAT1 and PI3K/AKT/NF-κB pathways to exacerbate the inflammatory phenotype in rheumatoid arthritis synovial fibroblasts and mouse embryonic fibroblasts".Arthritis Research and Therapy 20.1(2018).

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