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	ATG16L1 phosphorylation is oppositely regulated by CSNK2/casein kinase 2 and PPP1/protein phosphatase 1 which determines the fate of cardiomyocytes during hypoxia/reoxygenation
	Song, Huiwen[1,2]; Pu, Jun[2]; Wang, Lin[2]; Wu, Lihua[3]; Xiao, Jianmin[4]; Liu, Qigong[2]; Chen, Jun[5]; Zhang, Min[6]; Liu, Yang[7]; Ni, Mingke[8]
	2015
卷号	11期号:8页码:1308
URL标识	查看原文
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/4296074
专题	暨南大学
作者单位	1.[1]Southern Med Univ, Affiliated Baoan Hosp, Dept Cardiol, Shenzhen, Peoples R China 2.[2]Huazhong Univ Sci & Technol, Tongji Med Coll, Tongji Hosp, Dept Cardiol, Wuhan 430074, Peoples R China 3.[3]Zhejiang Univ, Affiliated Hosp 1, State Key Lab Diag & Treatment Infect Dis, Res Ctr Clin Pharm, Hangzhou 310003, Zhejiang, Peoples R China 4.[4]Jinan Univ, People Hosp Dongguan City 5, Guangzhou, Guangdong, Peoples R China 5.[5]Southern Med Univ, Affiliated Baoan Hosp, Dept Cardiol, Shenzhen, Peoples R China 6.[6]Huazhong Univ Sci & Technol, Tongji Med Coll, Union Hosp, Inst Cardiol, Wuhan 430074, Peoples R China 7.[7]Guangdong Acad Med Sci, Guangdong Cardiovasc Inst, Guangdong Gen Hosp, Guangzhou, Guangdong, Peoples R China 8.[8]Zhongshan Hosp, Dept Cardiol, Wuhan, Peoples R China 9.[9]Taizhou Cent Hosp, Taizhou, Peoples R China 10.[10]Hainan Prov Peoples Hosp, Haikou, Hainan, Peoples R China
推荐引用方式 GB/T 7714	Song, Huiwen[1,2],Pu, Jun[2],Wang, Lin[2],et al. ATG16L1 phosphorylation is oppositely regulated by CSNK2/casein kinase 2 and PPP1/protein phosphatase 1 which determines the fate of cardiomyocytes during hypoxia/reoxygenation[J],2015,11(8):1308.
APA	Song, Huiwen[1,2].,Pu, Jun[2].,Wang, Lin[2].,Wu, Lihua[3].,Xiao, Jianmin[4].,...&Zhang, Zhiyong[13,15].(2015).ATG16L1 phosphorylation is oppositely regulated by CSNK2/casein kinase 2 and PPP1/protein phosphatase 1 which determines the fate of cardiomyocytes during hypoxia/reoxygenation.,11(8),1308.
MLA	Song, Huiwen[1,2],et al."ATG16L1 phosphorylation is oppositely regulated by CSNK2/casein kinase 2 and PPP1/protein phosphatase 1 which determines the fate of cardiomyocytes during hypoxia/reoxygenation".11.8(2015):1308.

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