Upregulation of Transient Receptor Potential Canonical Channels Contributes to Endotoxin-Induced Pulmonary Arterial Stenosis | |
Chen, Gui-Lan; Jiang, Hongni; Zou, Fangdong | |
刊名 | MEDICAL SCIENCE MONITOR |
2016 | |
卷号 | 22 |
关键词 | Endotoxins Gene Expression Pulmonary Artery TRPC Cation Channels |
ISSN号 | 1643-3750 |
URL标识 | 查看原文 |
语种 | 英语 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/4284840 |
专题 | 复旦大学上海医学院 |
推荐引用方式 GB/T 7714 | Chen, Gui-Lan,Jiang, Hongni,Zou, Fangdong. Upregulation of Transient Receptor Potential Canonical Channels Contributes to Endotoxin-Induced Pulmonary Arterial Stenosis[J]. MEDICAL SCIENCE MONITOR,2016,22. |
APA | Chen, Gui-Lan,Jiang, Hongni,&Zou, Fangdong.(2016).Upregulation of Transient Receptor Potential Canonical Channels Contributes to Endotoxin-Induced Pulmonary Arterial Stenosis.MEDICAL SCIENCE MONITOR,22. |
MLA | Chen, Gui-Lan,et al."Upregulation of Transient Receptor Potential Canonical Channels Contributes to Endotoxin-Induced Pulmonary Arterial Stenosis".MEDICAL SCIENCE MONITOR 22(2016). |
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