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	TET1-GPER-PI3K/AKT pathway is involved in insulin-driven endometrial cancer cell proliferation
	Xie, Bing-ying; Lv, Qiao-ying; Ning, Cheng-cheng; Yang, Bing-yi; Shan, Wei-wei; Cheng, Ya-li; Gu, Chao; Luo, Xue-zhen; Zhang, Zhen-bo; Chen, Xiao-jun
刊名	BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
	2017
卷号	482期号:4
关键词	Insulin resistance Endometrial cancer GPER PI3K/AKT TETI
ISSN号	0006-291X
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3617496
专题	复旦大学上海医学院
推荐引用方式 GB/T 7714	Xie, Bing-ying,Lv, Qiao-ying,Ning, Cheng-cheng,et al. TET1-GPER-PI3K/AKT pathway is involved in insulin-driven endometrial cancer cell proliferation[J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS,2017,482(4).
APA	Xie, Bing-ying.,Lv, Qiao-ying.,Ning, Cheng-cheng.,Yang, Bing-yi.,Shan, Wei-wei.,...&Feng, You-ji.(2017).TET1-GPER-PI3K/AKT pathway is involved in insulin-driven endometrial cancer cell proliferation.BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS,482(4).
MLA	Xie, Bing-ying,et al."TET1-GPER-PI3K/AKT pathway is involved in insulin-driven endometrial cancer cell proliferation".BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 482.4(2017).

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