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	Inhibition of Rac1 ameliorates neuronal oxidative stress damage via reducing Bcl-2/Rac1 complex formation in mitochondria through PI3K/Akt/mTOR pathway
	Pan, Yundan; Wang, Na; Xia, Pingping; Wang, E.; Guo, Qulian; Ye, Zhi*
刊名	Experimental Neurology
	2018
卷号	300页码:149-166
关键词	Cerebral ischemia Hyperglycemia Oxidative stress Rac1 Neuroprotection Phosphatidylinositol 3-kinase (PI3K)
ISSN号	0014-4886
DOI	10.1016/j.expneurol.2017.10.030
URL标识	查看原文
WOS记录号	WOS:000422893300015
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3339383
专题	中南大学
作者单位	1.[Pan, Yundan 2.Guo, Qulian 3.Xia, Pingping 4.Wang, E. 5.Ye, Zhi 6.Wang, Na] Cent S Univ, Affiliated Xiangya Hosp, Dept Anesthesiol, Changsha 410078, Hunan, Peoples R China.
推荐引用方式 GB/T 7714	Pan, Yundan,Wang, Na,Xia, Pingping,et al. Inhibition of Rac1 ameliorates neuronal oxidative stress damage via reducing Bcl-2/Rac1 complex formation in mitochondria through PI3K/Akt/mTOR pathway[J]. Experimental Neurology,2018,300:149-166.
APA	Pan, Yundan,Wang, Na,Xia, Pingping,Wang, E.,Guo, Qulian,&Ye, Zhi*.(2018).Inhibition of Rac1 ameliorates neuronal oxidative stress damage via reducing Bcl-2/Rac1 complex formation in mitochondria through PI3K/Akt/mTOR pathway.Experimental Neurology,300,149-166.
MLA	Pan, Yundan,et al."Inhibition of Rac1 ameliorates neuronal oxidative stress damage via reducing Bcl-2/Rac1 complex formation in mitochondria through PI3K/Akt/mTOR pathway".Experimental Neurology 300(2018):149-166.

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