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Hypermethylation of DDAH2 promoter contributes to the dysfunction of endothelial progenitor cells in coronary artery disease patients
Niu, Pan-Pan; Cao, Yu; Gong, Ting; Guo, Jin-Hui; Zhang, Bi-Kui*; Jia, Su-Jie
刊名Journal of Translational Medicine
2014
卷号12期号:1页码:170
关键词Atherosclerosis DNA methylation Dimethylarginine dimethylaminohydrolase Endothelial progenitor cells Homocysteine
ISSN号1479-5876
DOI10.1186/1479-5876-12-170
URL标识查看原文
WOS记录号WOS:000338473900001
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3331050
专题中南大学
作者单位1.[Zhang, Bi-Kui
2.Niu, Pan-Pan
3.Jia, Su-Jie
4.Gong, Ting] Cent S Univ, Xiangya Hosp 3, Dept Pharmaceut, Changsha 410013, Hunan, Peoples R China.
推荐引用方式
GB/T 7714
Niu, Pan-Pan,Cao, Yu,Gong, Ting,et al. Hypermethylation of DDAH2 promoter contributes to the dysfunction of endothelial progenitor cells in coronary artery disease patients[J]. Journal of Translational Medicine,2014,12(1):170.
APA Niu, Pan-Pan,Cao, Yu,Gong, Ting,Guo, Jin-Hui,Zhang, Bi-Kui*,&Jia, Su-Jie.(2014).Hypermethylation of DDAH2 promoter contributes to the dysfunction of endothelial progenitor cells in coronary artery disease patients.Journal of Translational Medicine,12(1),170.
MLA Niu, Pan-Pan,et al."Hypermethylation of DDAH2 promoter contributes to the dysfunction of endothelial progenitor cells in coronary artery disease patients".Journal of Translational Medicine 12.1(2014):170.
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