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β‐Secretase‐1 elevation in transgenic mouse models of Alzheimer’s disease is associated with synaptic/axonal pathology and amyloidogenesis: implications for neuritic plaque development
Zhang, Xue-Mei; Cai, Yan; Xiong, Kun; Cai, Huaibin; Luo, Xue-Gang; Feng, Jia-Chun; Clough, Richard W.; Struble, Robert G.; Patrylo, Peter R.; Yan, Xiao-Xin*
刊名European Journal of Neuroscience
2009
卷号30期号:12页码:2271-2283
关键词aging axonopathy dystrophic neurite neuroplasticity secretase senile plaques
ISSN号1460-9568
DOI10.1111/j.1460-9568.2009.07017.x
URL标识查看原文
WOS记录号WOS:000272836200005
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3315458
专题中南大学
作者单位1.[Zhang, Xue-Mei
2.Yan, Xiao-Xin
3.Clough, Richard W.
4.Patrylo, Peter R.] So Illinois Univ, Sch Med, Dept Anat, Carbondale, IL 62901 USA.
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GB/T 7714
Zhang, Xue-Mei,Cai, Yan,Xiong, Kun,et al. β‐Secretase‐1 elevation in transgenic mouse models of Alzheimer’s disease is associated with synaptic/axonal pathology and amyloidogenesis: implications for neuritic plaque development[J]. European Journal of Neuroscience,2009,30(12):2271-2283.
APA Zhang, Xue-Mei.,Cai, Yan.,Xiong, Kun.,Cai, Huaibin.,Luo, Xue-Gang.,...&Yan, Xiao-Xin*.(2009).β‐Secretase‐1 elevation in transgenic mouse models of Alzheimer’s disease is associated with synaptic/axonal pathology and amyloidogenesis: implications for neuritic plaque development.European Journal of Neuroscience,30(12),2271-2283.
MLA Zhang, Xue-Mei,et al."β‐Secretase‐1 elevation in transgenic mouse models of Alzheimer’s disease is associated with synaptic/axonal pathology and amyloidogenesis: implications for neuritic plaque development".European Journal of Neuroscience 30.12(2009):2271-2283.
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