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	Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis
	Niu Ningning; Lu Ping; Yang Yanlin; He Ruizhe; Zhang Li; Shi Juanjuan; Wu Jinghua; Yang Minwei; Zhang Zhi-Gang; Wang Li-Wei
刊名	Gut
	2019
关键词	cancer genetics,pancreatic cancer
ISSN号	1468-3288
URL标识	查看原文
WOS记录号	[DB:DC_IDENTIFIER_WOSID]
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3230965
专题	大连理工大学
作者单位	1.State Key Laboratory of Oncogenes and Related Genes, Renji-Med X Clinical Stem Cell Research Center, Shanghai Cancer Institute, Shanghai Jiao Tong University School of Medicine Affiliated Renji Hospital, Shanghai, China. 2.Department of Biliary-Pancreatic Surgery, Shanghai Jiao Tong University School of Medicine Affiliated Renji Hospital, Shanghai, China. 3.Department of Oncology, Shanghai Jiao Tong University School of Medicine Affiliated Renji Hospital, Shanghai, China. 4.School of pharmaceutical Science and Technology, Dalian University of Technology, Dalian, China.
推荐引用方式 GB/T 7714	Niu Ningning,Lu Ping,Yang Yanlin,et al. Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis[J]. Gut,2019.
APA	Niu Ningning.,Lu Ping.,Yang Yanlin.,He Ruizhe.,Zhang Li.,...&Xue Jing.(2019).Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis.Gut.
MLA	Niu Ningning,et al."Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis".Gut (2019).

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