IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice. | |
Zhang Lisha; Liu Peining; Wen Wen; Bai Xiaofang; Zhang Yan; Liu Mengping; Wang Lijun; Wu Yue; Yuan Zuyi; Zhou Juan | |
刊名 | Molecular immunology |
2019 | |
卷号 | 105页码:240-250 |
关键词 | AMI IL-17A AMPK&alpha Inflammasome Macrophage |
ISSN号 | 1872-9142 |
URL标识 | 查看原文 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/2828420 |
专题 | 西安交通大学 |
推荐引用方式 GB/T 7714 | Zhang Lisha,Liu Peining,Wen Wen,et al. IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice.[J]. Molecular immunology,2019,105:240-250. |
APA | Zhang Lisha.,Liu Peining.,Wen Wen.,Bai Xiaofang.,Zhang Yan.,...&Zhou Juan.(2019).IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice..Molecular immunology,105,240-250. |
MLA | Zhang Lisha,et al."IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice.".Molecular immunology 105(2019):240-250. |
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