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Celastrol protects mouse retinas from bright light-induced degeneration through inhibition of oxidative stress and inflammation
Bian Minjuan[1]; Du Xiaoye[2]; Cui Jingang[3]; Wang Peiwei[4]; Wang Wenjian[5]; Zhu Weiliang[6]; Zhang Teng[7]; Chen Yu[8]
刊名Journal of neuroinflammation
2016
卷号13页码:50
ISSN号1742-2094
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2236451
专题上海大学
作者单位1.[1]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China.
2.[2]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China.
3.[3]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China.
4.[4]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China.
5.[5]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China.
6.[6]Shanghai Institute of Materia Medica, Shanghai, 201203, China.
7.[7]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China. zhangteng501@hotmail.com.
8.[8]Clinical Research Institute of Integrative Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai, 200437, China. chenyu6639@hotmail.com.
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GB/T 7714
Bian Minjuan[1],Du Xiaoye[2],Cui Jingang[3],et al. Celastrol protects mouse retinas from bright light-induced degeneration through inhibition of oxidative stress and inflammation[J]. Journal of neuroinflammation,2016,13:50.
APA Bian Minjuan[1].,Du Xiaoye[2].,Cui Jingang[3].,Wang Peiwei[4].,Wang Wenjian[5].,...&Chen Yu[8].(2016).Celastrol protects mouse retinas from bright light-induced degeneration through inhibition of oxidative stress and inflammation.Journal of neuroinflammation,13,50.
MLA Bian Minjuan[1],et al."Celastrol protects mouse retinas from bright light-induced degeneration through inhibition of oxidative stress and inflammation".Journal of neuroinflammation 13(2016):50.
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