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Heat shock protein gp96 decreases p53 stability by regulating Mdm2 E3 ligase activity in liver cancer.
Ye,Xin; Liu,Ningning; Wu,Bo; Hou,Junwei; Meng,Songdong; Li,Changfei; Fan,Hongxia; Chu,Xiaoyu; Kong,Xianping; Feng,Cong
刊名Cancer Letters
2015
卷号Vol.359 No.2页码:325-334
关键词TOLL-LIKE RECEPTORS HEPATOCELLULAR-CARCINOMA CHAPERONE GP96 THERAPEUTIC-TARGET MACROPHAGES METASTASIS GP96/GRP94 EXPRESSION ONCOGENE PATHWAY
ISSN号0304-3835
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2156254
专题安徽大学
作者单位1.458th Hosp, Ctr Infect Dis, Transgen Engn Res Lab, Guangzhou, Guangdong, Peoples R China
2.Chinese Acad Sci, Inst Microbiol, CAS Key Lab Pathogen Microbiol & Immunol, Beijing, Peoples R China
3.Anhui Univ, Sch Life Sci, Hefei 230039, Peoples R China
推荐引用方式
GB/T 7714
Ye,Xin,Liu,Ningning,Wu,Bo,et al. Heat shock protein gp96 decreases p53 stability by regulating Mdm2 E3 ligase activity in liver cancer.[J]. Cancer Letters,2015,Vol.359 No.2:325-334.
APA Ye,Xin.,Liu,Ningning.,Wu,Bo.,Hou,Junwei.,Meng,Songdong.,...&Feng,Cong.(2015).Heat shock protein gp96 decreases p53 stability by regulating Mdm2 E3 ligase activity in liver cancer..Cancer Letters,Vol.359 No.2,325-334.
MLA Ye,Xin,et al."Heat shock protein gp96 decreases p53 stability by regulating Mdm2 E3 ligase activity in liver cancer.".Cancer Letters Vol.359 No.2(2015):325-334.
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