| 铜离子对中国花鲈幼鱼的毒性研究; Toxicity of Cu--(2+) to Juvenile Perch Lateolabrax maculatus | |
| 朱友芳 ; 洪万树 ; 林金忠 | |
| 2011 | |
| 关键词 | 中国花鲈 铜离子 半致死浓度 肝脏组织 酶活力 微核测定 Lateolabrax maculates Cu2+ LC50 liver tissue enzyme activity micronuclear test |
| 英文摘要 | 为给中国花鲈(lATEOlAbrAX MACulATuS)养殖提供污染生物学的理论数据,研究了中国花鲈幼鱼的铜离子中毒症状和半致死浓度(lC50);检测了幼鱼肝脏的谷胱甘肽(gSH)和丙二醛(MdA)含量及超氧化物歧化酶(SOd)、过氧化氢酶(CAT)、谷丙转氨酶(gPT)和谷草转氨酶(gOT)活力;分析了铜离子胁迫对幼鱼的红细胞总核异率和微核率的影响。结果表明:铜离子对幼鱼24、48、72和96H的lC50分别为1.85、1.66、1.48和0.86Mg·l-1。濒死的幼鱼肝脏组织溶解性坏死,鳃部组织出现大量的血窦,幼鱼肝脏的gSH和MdA含量及SOd、gPT和gOT活力对铜离子胁迫反应敏感,而CAT活力则反应不敏感。在铜离子浓度为0.42Mg·l-1的浓度组中暴露96H,幼鱼的总核异率和微核率显著高于对照组(P<0.05)。以上结果说明铜离子胁迫损害了中国花鲈幼鱼肝脏的抗氧化系统、肝脏组织和鳃部组织,并具有一定的遗传毒性。; In order to provide data of pollution biology for the perch(Lateolabrax maculatus) culture,the toxic symptom and LC50 were studied in the juvenile perch exposed to Cu2+.The levels of GSH and MDA and activities of SOD,CAT,GOT and GPT were examined;The effects of Cu2+ stress on the total dyskaryosis and micronuclear rates of the juvenile erythrocytes were analyzed.The results showed that LC50 for juvenile exposed to Cu2+ for 24,48,72 and 96 h was 1.85,1.66,1.48 and 0.86 mg·L-1,respectively.The liver tissue of agonal individuals necrosed and a mass of blood sinuses occurred in the gill tissue.GSH,MDA,SOD,GPT and GOT in the liver were sensitive,while CAT was not sensitive to Cu2+ stress.Exposed to Cu2+ at concentration of 0.42 mg L-1 for 96 h,total dyskaryosis and micronuclear rates of the juvenile erythrocytes in the treated groups were significantly higher(p<0.05) than that in the control.Cu2+ stress not only damaged the liver antioxidant system,liver and gill tissues,but also had definite genetic toxity to the juvenile perch.; 福建省莆田市科技局项目[2009N15(5)] |
| 语种 | zh_CN |
| 内容类型 | 期刊论文 |
| 源URL | [http://dspace.xmu.edu.cn/handle/2288/104409]  |
| 专题 | 海洋环境－已发表论文 |
| 推荐引用方式 GB/T 7714 | 朱友芳,洪万树,林金忠. 铜离子对中国花鲈幼鱼的毒性研究, Toxicity of Cu--(2+) to Juvenile Perch Lateolabrax maculatus[J],2011. |
| APA | 朱友芳,洪万树,&林金忠.(2011).铜离子对中国花鲈幼鱼的毒性研究.. |
| MLA | 朱友芳,et al."铜离子对中国花鲈幼鱼的毒性研究".(2011). |
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