Ca2+ signaling initiated by canonical transient receptor potential channels in dendritic development | |
Feng, SJ; He, ZH; Li, HY; Wang, YZ | |
刊名 | NEUROSCIENCE BULLETIN |
2015 | |
卷号 | 31期号:3页码:351-356 |
关键词 | NEURITE OUTGROWTH TRPC CHANNELS NEUROTROPHIC FACTORS HIPPOCAMPAL-NEURONS CATION CHANNEL GROWTH CONES BRAIN CELLS BDNF NEUROBLASTS |
通讯作者 | Wang, YZ (reprint author), Shanghai Inst Biol Sci, State Key Lab Neurosci, Inst Neurosci, Lab Neural Signal Transduct, Shanghai 200031, Peoples R China.,yzwang@ion.ac.cn |
英文摘要 | The spatial patterns of dendritic structures diverge in different types of neurons as adaptations to their unique functions. Although different intracellular mechanisms underlying dendritic morphogenesis have been suggested, it is evident that the elevation in intracellular Ca2+ levels plays a major role in the process. Canonical transient receptor potential (TRPC) channels, known to be non-selective Ca2+-permeable cation channels, act as environmental detectors to sense and transduce extracellular signals into different intracellular responses, including the regulation of dendritic growth, via Ca2+ influx. Here, we review recent advances in the understanding of Ca2+ signaling, especially signals mediated by Ca2+ influx via TRPC channels, and the underlying molecular events in dendritic development. |
学科主题 | Neurosciences |
WOS标题词 | Neurosciences & Neurology |
收录类别 | SCI |
语种 | 英语 |
WOS记录号 | WOS:000355619700008 |
公开日期 | 2016-02-26 |
内容类型 | 期刊论文 |
源URL | [http://ir.sibs.ac.cn/handle/331001/3946] |
专题 | 上海神经科学研究所_神经所(总) |
推荐引用方式 GB/T 7714 | Feng, SJ,He, ZH,Li, HY,et al. Ca2+ signaling initiated by canonical transient receptor potential channels in dendritic development[J]. NEUROSCIENCE BULLETIN,2015,31(3):351-356. |
APA | Feng, SJ,He, ZH,Li, HY,&Wang, YZ.(2015).Ca2+ signaling initiated by canonical transient receptor potential channels in dendritic development.NEUROSCIENCE BULLETIN,31(3),351-356. |
MLA | Feng, SJ,et al."Ca2+ signaling initiated by canonical transient receptor potential channels in dendritic development".NEUROSCIENCE BULLETIN 31.3(2015):351-356. |
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